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GSH and its precursors (L-cysteine and N-acetyl cysteine) treatments restore epigenetic modifying enzymes and VD metabolism genes Supplementation of hepatocytes with GSH or its precursors does not affect cell viability

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mansoni TGR was first cloned, we proposed that the enzyme was a selenoprotein with a penultimate Sec residue, and that TGR should have enzymatic activity dependent on the C-terminal Sec-containing motif in analogy to mammalian TrxR and TGR isoforms [11]