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Background A biopharmaceutical company was developing a novel ADC with a core requirement to design a chemically cleavable linker capable of specifically releasing cytotoxins inside tumor cells
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Indeed, in both mammalian and Drosophila cells, overexpression of GTP-bound mutants of RagA or RagB renders mTORC1 constitutively active and insensitive to amino acid starvation [26,54]
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