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These experiments indicated that, in addition to requiring Cys-dependent de novo biosynthesis of GSH for survival (see above), TrxR1/Gsr-null livers require de novo biosynthesis of Cys via transsulfuration, and that they cannot effectively utilize cystine as a hepatic Cys source [107]
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A recent study in Sri Lanka analyzed the association between the GPX1 rs1050450 Pro198Leu variant and coronary artery disease and reported that the variant was increased in patients compared to controls (OR, 2.84
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