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10.4236/ojmip.2013.33019 150 MazeI.NestlerE

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ECs contribute to sepsis pathogenesis through the activation of intracellular inflammatory pathways mediated by nuclear factor kappa B (NF-κB) and mitogen-activated protein kinase (MAPK), which are dependent on Toll-like receptors (TLR) ( Furthermore, copper overload damages T- and B-lymphocytes, leading to immunosuppression

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