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MiR-23b and miR-130b were reported to be downregulated and to inhibit cell growth in pituitary adenomas (58)
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At progression, the metabolic profile indicates chronic BM niche remodeling: sustained CD38 blockade only partially limits ADO generation above immunoregulatory thresholds, while increased ADA/CD26 activity accelerates ADO deamination and shifts purine flux toward INO
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