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Macrophages in diabetic environments show distinct morphological features of ferroptosis and altered expression of related molecules, leading to persistent and aggravated inflammation [76]

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Moreover, the provision of IL-15 and IGF-1 to CD34 + cells was associated with upregulated mRNA signals for PRF1 (which encodes perforin), GzmB (which encodes Granzyme B) and IFNG (which encodes interferon-γ (IFN-γ)) (Fig