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This imbalance results in the overproduction of pro-inflammatory cytokines, further exacerbating mucosal damage [7, 41]
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It has been found that curculigoside improves the sensitivity of intestinal epithelial cell GPx4 to selenium, promotes GPx4 expression, and alleviates histological damage of the colon in UC mice induced by dextran sulfate sodium (DSS) [278]
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