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Interestingly, the suppression of ER stress by VD was accompanied by upregulated M1 macrophages and stimulated ER stress promoted higher levels of “M2 macrophages” which were beneficial to foam cell formation and atherosclerosis plaque development [139–141]

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The inhibition of HMGCR was associated with decreased triglycerides and LDL levels in plasma [91]

Disodium EDTA