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Based on the findings presented here, we predict that one of these sequelae is an increased sensitization to ferroptosis downstream of diminished levels of neuronal selenium and GPX4
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Gln is metabolized by GLS to produce alpha-ketoglutaric acid (α-KG), which serves as a crucial cofactor for TET enzymes that catalyze DNA demethylation, as well as for histone demethylases like JHDM
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