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A study analyzing data from the RPE/choroid and neurosensory retina of AMD patients, revealed that hypermethylation of the GSTM1 and GSTM5 promoters compromised the antioxidant defense of retinal cells
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in its active form (thiamine diphosphate), it is an essential cofactor for enzymes in glucose and amino acid catabolism, helping convert nutrients into ATP and supporting normal neuronal function
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