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Our data also indicated that C18:3 was present in both depleted and enriched TAGs in T2DM [55]
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Propyl hydroxylase function requires iron and inactivating mutations in propyl hydroxylases result in increased HIF concentration and induction of erythrocytosis, overriding the upregulation of hepcidin associated with inflammation [89], resulting in hepcidin suppression [90]
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