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Antibody drugs may also selectively interfere with B-cell activity, reducing antibody production and the release of inflammatory mediators
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Regarding therapeutic interventions for adipose tissue fibrosis, the phytochemical Sulforaphane has been shown to mitigate fibrosis by promoting an M2-polarized macrophage phenotype via activation of the Nrf2 pathway (194), while exercise training can attenuate diet-induced WAT fibrosis in obese mice by reducing TGF-β levels and macrophage infiltration (195)
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