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Equally critical is the identification of biomarkers capable of defining ASD subgroups in whom intervention with oxytocin, a synthetic analog or even an antagonist of OXTR, might yield beneficial outcomes, given the heterogeneity in ASD etiology, phenotype, and treatment response [112, 113]
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Moreover, lymphocyte proliferation, activation, and differentiation, as well as cytokine production, were the most affected gene ontology categories in cecal tonsils (151)
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