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NAD metabolism fuels human and mouse intestinal inflammation

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Potency:100 mg

The deletion constructs of CTD (cFSP1 ΔCTD , residues 1–318, or hFSP1 ΔCTD , residues 1–316) abolished the NADH oxidation in vitro and hFSP1 ΔCTD obviously decreased the ferroptosis inhibition activities in cells (Fig

which protects against lipid peroxidation (fats turning rancid).